INT306831

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Context Info
Confidence 0.10
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.46
Pain Relevance 0.85

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (E2F4, NOLC1) nucleus (E2F4, NOLC1) DNA binding (E2F4, NOLC1)
transcription factor binding (E2F4, NOLC1) mitosis (NOLC1) nucleolus (NOLC1)
E2F4 (Homo sapiens)
NOLC1 (Homo sapiens)
Pain Link Frequency Relevance Heat
qutenza 130 99.98 Very High Very High Very High
Inflammation 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Small Cell Lung Cancer 63 75.00 Quite High
Cancer 42 74.28 Quite High
Disease 3 50.32 Quite High
Lung Cancer 10 5.00 Very Low Very Low Very Low
Apoptosis 6 5.00 Very Low Very Low Very Low
Reprotox - General 1 4 5.00 Very Low Very Low Very Low
Metastasis 2 5.00 Very Low Very Low Very Low
Weight Loss 2 5.00 Very Low Very Low Very Low
Breast Cancer 2 5.00 Very Low Very Low Very Low
Non-small-cell Lung Cancer 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
ChIP assays demonstrated that capsaicin caused the recruitment of E2F4 and p130 on E2F-responsive proliferative promoters, thereby inhibiting cell proliferation.


Positive_regulation (caused) of E2F4 Binding (recruitment) of p130 associated with qutenza
1) Confidence 0.10 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2857654 Disease Relevance 0.46 Pain Relevance 0.85

General Comments

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