INT306921

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Context Info
Confidence 0.15
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.79
Pain Relevance 0.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Ang) Golgi apparatus (Ephx2) peroxisome (Ephx2)
cytoplasm (Ephx2) nuclease activity (Ang) cytosol (Ephx2)
Anatomy Link Frequency
AT-1 2
Ang (Mus musculus)
Ephx2 (Mus musculus)
Pain Link Frequency Relevance Heat
antagonist 2 95.04 Very High Very High Very High
Inflammation 24 82.48 Quite High
cytokine 4 61.12 Quite High
Pain 2 45.56 Quite Low
IPN 1 30.36 Quite Low
Analgesic 1 21.84 Low Low
Potency 5 5.00 Very Low Very Low Very Low
cva 3 5.00 Very Low Very Low Very Low
aspirin 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypertension 14 97.00 Very High Very High Very High
Coronary Heart Disease 8 93.80 High High
Natriuresis 1 86.48 High High
INFLAMMATION 25 82.48 Quite High
Myocardial Infarction 4 81.68 Quite High
Diabetes Mellitus 3 46.44 Quite Low
Stroke 9 45.88 Quite Low
Pain 2 45.56 Quite Low
Atherosclerosis 53 45.32 Quite Low
Disease 5 43.96 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This study revealed that Ang II dose-dependently upregulated cardiac sEH expression involving AP-1 transcription factor activation, which could be reversed by losartan, an angiotensin type 1 (AT-1) receptor antagonist.
Ang Positive_regulation (upregulated) of Gene_expression (expression) of sEH in AT-1 associated with antagonist
1) Confidence 0.15 Published 2010 Journal Curr Atheroscler Rep Section Body Doc Link PMC2857794 Disease Relevance 0.79 Pain Relevance 0.12

General Comments

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