INT308158

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Context Info
Confidence 0.08
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.18
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (CPT1B) mitochondrion (CPT1B) small molecule metabolic process (CPT1B)
transferase activity, transferring acyl groups (CPT1B)
CPT1B (Homo sapiens)
Pain Link Frequency Relevance Heat
Bioavailability 5 28.24 Quite Low
Inflammation 5 5.00 Very Low Very Low Very Low
Pain 5 5.00 Very Low Very Low Very Low
ischemia 4 5.00 Very Low Very Low Very Low
Inflammatory response 4 5.00 Very Low Very Low Very Low
diabetic neuropathy 4 5.00 Very Low Very Low Very Low
cytokine 3 5.00 Very Low Very Low Very Low
Neuropathic pain 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Acidosis 1 97.68 Very High Very High Very High
Injury 9 82.56 Quite High
Body Weight 4 20.08 Low Low
Syndrome 4 8.16 Low Low
Disease 22 5.00 Very Low Very Low Very Low
Appetite Loss 16 5.00 Very Low Very Low Very Low
Diabetes Mellitus 12 5.00 Very Low Very Low Very Low
Sepsis 10 5.00 Very Low Very Low Very Low
Cirrhosis 9 5.00 Very Low Very Low Very Low
Hepatic Encephalopathy 9 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
CPTI (Figure 2) is a control point of FA oxidation and decreased carnitine levels and acidosis of CPT1 have been implicated in decreased fatty acid oxidation during heavy exercise [18].
Regulation (control) of CPTI associated with acidosis
1) Confidence 0.08 Published 2010 Journal Nutr Metab (Lond) Section Body Doc Link PMC2861661 Disease Relevance 0.18 Pain Relevance 0

General Comments

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