INT308854

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Context Info
Confidence 0.14
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Bcl2a1a, HTT) Golgi apparatus (HTT) endoplasmic reticulum (HTT)
protein complex (HTT) cytosol (HTT) cell death (HTT)
Anatomy Link Frequency
primary neurons 2
Bcl2a1a (Mus musculus)
HTT (Homo sapiens)
Pain Link Frequency Relevance Heat
Clonidine 7 5.00 Very Low Very Low Very Low
Hippocampus 3 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
medulla 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
cerebral cortex 1 5.00 Very Low Very Low Very Low
antinociception 1 5.00 Very Low Very Low Very Low
nMDA receptor 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Antinociceptive 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Toxicity 3 50.00 Quite Low
Weight Loss 4 47.92 Quite Low
Anorexia 1 44.32 Quite Low
Tremor 11 20.48 Low Low
Disease 50 5.00 Very Low Very Low Very Low
Targeted Disruption 19 5.00 Very Low Very Low Very Low
Death 7 5.00 Very Low Very Low Very Low
Hypertension 4 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 4 5.00 Very Low Very Low Very Low
Sleep Disorders 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The stable inducible PC12 cells have been previously described (11), cells were treated with doxycycline to induce huntingtin expression for 24 h at 1 µg/ml with the addition of 1 µm rilmenidine for a further 24 h and bafilomycin A1 as for primary neurons.


A1 Positive_regulation (induce) of Gene_expression (expression) of huntingtin in primary neurons
1) Confidence 0.14 Published 2010 Journal Human Molecular Genetics Section Body Doc Link PMC2865373 Disease Relevance 0 Pain Relevance 0

General Comments

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