INT309148

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.54
Pain Relevance 0.36

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Dlat) cell cycle (Cks2) molecular_function (Cks2)
transferase activity, transferring acyl groups (Dlat) kinase activity (Cks2) cellular_component (Cks2)
Anatomy Link Frequency
neurons 1
Dlat (Rattus norvegicus)
Cks2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 67 83.20 Quite High
bDMF 24 82.40 Quite High
Hippocampus 14 76.48 Quite High
antagonist 8 70.68 Quite High
agonist 2 40.92 Quite Low
anesthesia 4 5.00 Very Low Very Low Very Low
cerebral cortex 1 5.00 Very Low Very Low Very Low
Pyramidal cell 1 5.00 Very Low Very Low Very Low
halothane 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cv General 4 Under Development 59 83.20 Quite High
Death 6 71.24 Quite High
Apoptosis 5 69.80 Quite High
Cv Unclassified Under Development 6 63.96 Quite High
Middle Cerebral Artery Infarction 3 63.16 Quite High
Targeted Disruption 9 29.68 Quite Low
Cognitive Disorder 16 5.00 Very Low Very Low Very Low
Stroke 6 5.00 Very Low Very Low Very Low
Brain Hemorrhage 2 5.00 Very Low Very Low Very Low
Disease 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Along these lines, several laboratories have shown that the rapid activation of extracellular signal-regulated kinases 1,2 (ERKs) by E2 is critical for its neuroprotective effects, as administration of a MEK inhibitor blocks E2 neuroprotection in neurons in vitro [24], [25], [26].
E2 Positive_regulation (activation) of kinases 1,2 in neurons
1) Confidence 0.00 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2866326 Disease Relevance 0.54 Pain Relevance 0.36

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox