INT309353

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Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0.90

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

oxidoreductase activity (Glyr1) nucleus (Glyr1) DNA binding (Glyr1)
cellular_component (Glyr1)
Anatomy Link Frequency
neurons 4
Glyr1 (Mus musculus)
Pain Link Frequency Relevance Heat
antagonist 10 98.64 Very High Very High Very High
gABA 56 95.24 Very High Very High Very High
Neurotransmitter 60 95.04 Very High Very High Very High
Spinal cord 96 76.56 Quite High
agonist 8 54.64 Quite High
nMDA receptor 6 53.88 Quite High
interneuron 2 49.12 Quite Low
GABAergic 34 25.36 Quite Low
midbrain 16 5.00 Very Low Very Low Very Low
Hippocampus 14 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 20 5.00 Very Low Very Low Very Low
Pain 6 5.00 Very Low Very Low Very Low
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Nociception 4 5.00 Very Low Very Low Very Low
Sleep Disorders 2 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Lung Cancer 2 5.00 Very Low Very Low Very Low
Spasticity 2 5.00 Very Low Very Low Very Low
Depression 2 5.00 Very Low Very Low Very Low
Muscle Hypertonia 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Experiments using the GlyR antagonist strychnine and L-type Ca+2 blockers on spinal neurons suggest that GlyR activation leads to Ca+2 transients that in turn cause accumulation of the anchoring protein Gephyrin and GlyR.
Positive_regulation (cause) of Positive_regulation (accumulation) of GlyR in neurons associated with antagonist
1) Confidence 0.03 Published 2010 Journal Frontiers in Molecular Neuroscience Section Body Doc Link PMC2866564 Disease Relevance 0 Pain Relevance 0.44
Experiments using the GlyR antagonist strychnine and L-type Ca+2 blockers on spinal neurons suggest that GlyR activation leads to Ca+2 transients that in turn cause accumulation of the anchoring protein Gephyrin and GlyR.
Positive_regulation (leads) of Positive_regulation (activation) of GlyR in neurons associated with antagonist
2) Confidence 0.03 Published 2010 Journal Frontiers in Molecular Neuroscience Section Body Doc Link PMC2866564 Disease Relevance 0 Pain Relevance 0.47

General Comments

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