INT309506

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Context Info
Confidence 0.23
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.19
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Tdp1) cytoplasm (Tdp1) nuclease activity (Tdp1)
Tdp1 (Mus musculus)
Pain Link Frequency Relevance Heat
cerebral cortex 6 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Hippocampus 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cerebellar Ataxia 1 98.44 Very High Very High Very High
Disease 50 97.20 Very High Very High Very High
Werner Syndrome 1 91.80 High High
Cockayne Syndrome 1 88.44 High High
Aging 6 87.04 High High
Neurodegenerative Disease 2 84.96 Quite High
Targeted Disruption 33 64.52 Quite High
Lifespan 5 59.68 Quite High
Dna Damage 4 41.84 Quite Low
Stress 5 21.88 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The autosomal recessive cerebellar ataxias, including AOA1/EAOH (Date et al., 2001), AOA2 (Suraweera et al., 2007), and SCAN1 (Takashima et al., 2002; El-Khamisy et al., 2005), are also caused by mutations of DNA repair genes.
Positive_regulation (caused) of SCAN1 associated with cerebellar ataxia
1) Confidence 0.23 Published 2010 Journal The Journal of Cell Biology Section Body Doc Link PMC2867301 Disease Relevance 1.19 Pain Relevance 0

General Comments

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