INT309738

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Context Info
Confidence 0.04
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (CALM3, CA2) cytosol (CALM3, CA2) nucleoplasm (CALM3)
lyase activity (CA2) small molecule metabolic process (CALM3) carbohydrate metabolic process (CALM3)
Anatomy Link Frequency
plasma 1
CALM3 (Homo sapiens)
CA2 (Homo sapiens)
Pain Link Frequency Relevance Heat
adenocard 2 82.60 Quite High
TRP channel 8 74.76 Quite High
agonist 6 5.00 Very Low Very Low Very Low
addiction 4 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
qutenza 2 5.00 Very Low Very Low Very Low

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
How does CaM binding facilitate Ca2+ entry through TRPV4 at the molecular level?
CaM Positive_regulation (facilitate) of Ca2
1) Confidence 0.04 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2867956 Disease Relevance 0 Pain Relevance 0.08
Prototypical mechanisms involving the displacement of autoinhibitory domains from a functional center are found in the CaM-dependent activation of CaMK II [39] and the plasma membrane Ca2+ pump [40], [41].
CaM Positive_regulation (activation) of Ca2 in plasma
2) Confidence 0.04 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2867956 Disease Relevance 0 Pain Relevance 0

General Comments

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