INT309839

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Context Info
Confidence 0.29
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 5
Disease Relevance 1.95
Pain Relevance 0.38

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell death (Nlrp1a) nucleus (Nlrp1a) enzyme binding (Nlrp1a)
Anatomy Link Frequency
liver 1
brain 1
Nlrp1a (Mus musculus)
Pain Link Frequency Relevance Heat
Paracetamol 4 99.48 Very High Very High Very High
Substantia nigra 60 91.32 High High
Dopamine 18 87.12 High High
medulla 5 5.00 Very Low Very Low Very Low
cytokine 4 5.00 Very Low Very Low Very Low
ketamine 3 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
Inflammatory response 3 5.00 Very Low Very Low Very Low
imagery 3 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Overdose 1 99.84 Very High Very High Very High
Hepatotoxicity 1 98.40 Very High Very High Very High
Targeted Disruption 78 94.56 High High
Deafness 18 91.20 High High
Stress 38 83.76 Quite High
Disease 84 83.68 Quite High
Toxicity 24 81.04 Quite High
Sprains And Strains 9 80.92 Quite High
Parkinson's Disease 12 72.36 Quite High
Death 1 6.64 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
L-NAC interacts directly with free radicals to prevent liver damage.
NAC Binding (interacts) of in liver associated with hepatotoxicity
1) Confidence 0.29 Published 2010 Journal J Occup Med Toxicol Section Body Doc Link PMC2868036 Disease Relevance 0.62 Pain Relevance 0.10
Chronic oral treatment with NAC increases the density of TH+ terminals in SNCA-overexpressing mice
NAC Binding (treatment) of
2) Confidence 0.06 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2925900 Disease Relevance 0.40 Pain Relevance 0
Despite the transient nature of the impact of NAC on brain glutathione, the loss of dopaminergic terminals at 1 year associated with SNCA overexpression was significantly attenuated by NAC supplementation, as measured by immunoreactivity for tyrosine hydroxylase in the striatum (p?
NAC Binding (supplementation) of in brain
3) Confidence 0.06 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2925900 Disease Relevance 0.58 Pain Relevance 0.15
Treatment with NAC Increases Cytosolic localization of NF?
NAC Binding (Treatment) of
4) Confidence 0.06 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2925900 Disease Relevance 0.35 Pain Relevance 0.13
For example, NAC, a representative antioxidant, was shown to inhibit TNF-?
NAC Binding (shown) of
5) Confidence 0.05 Published 2010 Journal Anatomy & Cell Biology Section Body Doc Link PMC2998772 Disease Relevance 0 Pain Relevance 0

General Comments

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