INT310250

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.68
Pain Relevance 0.37

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (TLE1) nucleus (TLE1) transcription factor binding (TLE1)
Anatomy Link Frequency
brain 1
TLE1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Cannabinoid 37 99.40 Very High Very High Very High
agonist 25 75.88 Quite High
antagonist 12 73.60 Quite High
Glutamate 26 53.72 Quite High
Neurotransmitter 1 25.16 Quite Low
Action potential 7 5.00 Very Low Very Low Very Low
Hippocampus 5 5.00 Very Low Very Low Very Low
GABAergic 5 5.00 Very Low Very Low Very Low
gABA 4 5.00 Very Low Very Low Very Low
tetrodotoxin 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Epilepsy 51 100.00 Very High Very High Very High
Sclerosis 4 92.32 High High
Convulsion 34 71.68 Quite High
Recurrence 1 71.20 Quite High
Status Epilepticus 37 15.56 Low Low
Gliosis 2 5.00 Very Low Very Low Very Low
Neurological Disease 1 5.00 Very Low Very Low Very Low
Primary Sclerosing Cholangitis 1 5.00 Very Low Very Low Very Low
Myoclonus 1 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, further study is needed to understand the interactions between cannabinoids and other receptor targets as well as cannabinoid effects in other brain areas affected in TLE or in other TLE models, where axon sprouting may be significant but not as robust as in the pilocarpine-treated mouse.



Regulation (affected) of TLE in brain associated with epilepsy and cannabinoid
1) Confidence 0.00 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2871782 Disease Relevance 0.68 Pain Relevance 0.37

General Comments

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