INT310488

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Context Info
Confidence 0.28
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.25
Pain Relevance 0.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Ascl1) nucleus (Ascl1)
Anatomy Link Frequency
interneurons 1
Ascl1 (Mus musculus)
Pain Link Frequency Relevance Heat
cerebral cortex 28 97.40 Very High Very High Very High
GABAergic 40 89.92 High High
interneuron 8 42.88 Quite Low
imagery 10 5.00 Very Low Very Low Very Low
Action potential 7 5.00 Very Low Very Low Very Low
antagonist 4 5.00 Very Low Very Low Very Low
Neurotransmitter 3 5.00 Very Low Very Low Very Low
Glutamate 3 5.00 Very Low Very Low Very Low
gABA 2 5.00 Very Low Very Low Very Low
Potency 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Infection 37 73.80 Quite High
Neurodegenerative Disease 18 57.12 Quite High
Injury 11 5.00 Very Low Very Low Very Low
Stab Wounds 5 5.00 Very Low Very Low Very Low
Targeted Disruption 3 5.00 Very Low Very Low Very Low
Stuttering 2 5.00 Very Low Very Low Very Low
Vesicular Stomatitis 2 5.00 Very Low Very Low Very Low
Brain Injury 2 5.00 Very Low Very Low Very Low
Epilepsy 1 5.00 Very Low Very Low Very Low
Dislocations 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Taken together, our data provide evidence that postnatal astroglia from the cerebral cortex can be driven towards the generation of interneurons with distinct functional properties by forced expression of Dlx2 or Dlx2 in combination with Mash1.


Mash1 Binding (combination) of in interneurons associated with cerebral cortex
1) Confidence 0.28 Published 2010 Journal PLoS Biology Section Body Doc Link PMC2872647 Disease Relevance 0.25 Pain Relevance 0.16

General Comments

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