INT310595

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Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.84
Pain Relevance 0.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Ros1, Npr1) cell differentiation (Ros1) cell proliferation (Ros1)
intracellular (Npr1)
Ros1 (Rattus norvegicus)
Npr1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 37 93.60 High High
anesthesia 4 74.08 Quite High
cytokine 1 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
potassium channel 1 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
Action potential 1 5.00 Very Low Very Low Very Low
isoflurane 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 19 100.00 Very High Very High Very High
Heart Disease 1 99.92 Very High Very High Very High
Death 4 98.20 Very High Very High Very High
Reperfusion Injury 14 96.36 Very High Very High Very High
Cv Unclassified Under Development 37 93.60 High High
Injury 4 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Metabolic Disorder 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low
Myocardial Infarction 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
ROS are now known to induce apoptosis and activate guanylate cyclase and transcriptional factors that act as secondary messengers, both mechanisms which result in cardiac cell death.
ROS Positive_regulation (activate) of guanylate cyclase associated with apoptosis and death
1) Confidence 0.03 Published 2010 Journal Korean Journal of Anesthesiology Section Body Doc Link PMC2872860 Disease Relevance 0.84 Pain Relevance 0.16

General Comments

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