INT311134

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Context Info
Confidence 0.27
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.73
Pain Relevance 0.10

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Calm2) nucleoplasm (Calm2) cytoskeleton (Calm2)
cell cycle (Calm2) cellular_component (Calm2) biological_process (Calm2)
Anatomy Link Frequency
neurons 2
Calm2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Pyramidal cell 24 83.00 Quite High
Inflammation 11 41.76 Quite Low
Hippocampus 17 7.68 Low Low
nMDA receptor 4 5.00 Very Low Very Low Very Low
chemokine 4 5.00 Very Low Very Low Very Low
Cannabinoid 4 5.00 Very Low Very Low Very Low
GABAergic 3 5.00 Very Low Very Low Very Low
long-term potentiation 2 5.00 Very Low Very Low Very Low
gABA 2 5.00 Very Low Very Low Very Low
ischemia 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Status Epilepticus 57 99.52 Very High Very High Very High
Toxicity 1 95.48 Very High Very High Very High
Death 2 95.04 Very High Very High Very High
Epilepsy 16 84.56 Quite High
Convulsion 18 61.92 Quite High
Gliosis 4 42.72 Quite Low
INFLAMMATION 12 41.76 Quite Low
Apoptosis 8 29.88 Quite Low
Injury 4 5.00 Very Low Very Low Very Low
Cv Unclassified Under Development 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The reduced CaM expression in our study is also interesting since there is extensive evidence that CaM plays a critical role in the induction of cell death following Ca2+ overload, and inhibition of CaM function can protect neurons from death [48], [49].
Negative_regulation (reduced) of Gene_expression (expression) of CaM in neurons associated with death
1) Confidence 0.27 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2873964 Disease Relevance 0.73 Pain Relevance 0.10

General Comments

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