INT311330

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Context Info
Confidence 0.35
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.85
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mRNA binding (Celf1) mRNA processing (Celf1) RNA binding (Celf1)
nucleus (Celf1) cytoplasm (Celf1)
Celf1 (Mus musculus)
Pain Link Frequency Relevance Heat
cytokine 4 5.00 Very Low Very Low Very Low
Central nervous system 4 5.00 Very Low Very Low Very Low
spastic colon 2 5.00 Very Low Very Low Very Low
depression 2 5.00 Very Low Very Low Very Low
Inflammation 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 38 91.08 High High
Frailty 14 86.00 High High
Arrhythmias 2 Under Development 4 80.96 Quite High
Myotonia 28 79.84 Quite High
Sprains And Strains 4 78.48 Quite High
Disease 26 58.56 Quite High
Myotonic Dystrophy 44 50.00 Quite Low
Toxicity 32 36.64 Quite Low
Cataract 14 30.84 Quite Low
Congenital Anomalies 22 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
More studies are also needed to determine the contribution of the reduction of MBNL1 and elevation of CUGBP1 in DM1 phenotype.
Negative_regulation (reduction) of Positive_regulation (elevation) of CUGBP1
1) Confidence 0.35 Published 2010 Journal Current Genomics Section Body Doc Link PMC2874224 Disease Relevance 0.37 Pain Relevance 0
Based on these results, it is reasonable to suggest that DM1 phenotype could be caused equally well by the reduction of MBNL1 and by the increase of CUGBP1.
Negative_regulation (reduction) of Positive_regulation (increase) of CUGBP1
2) Confidence 0.34 Published 2010 Journal Current Genomics Section Body Doc Link PMC2874224 Disease Relevance 0.48 Pain Relevance 0

General Comments

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