INT311535

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Context Info
Confidence 0.19
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.61
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (CA2) cell differentiation (ROS1) cell proliferation (ROS1)
extracellular space (CA2) lyase activity (CA2) plasma membrane (ROS1)
ROS1 (Homo sapiens)
CA2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Central nervous system 11 76.96 Quite High
Ventral tegmentum 9 33.04 Quite Low
Neurotransmitter 3 28.72 Quite Low
Glutamate 17 23.16 Low Low
Substantia nigra 12 5.00 Very Low Very Low Very Low
ischemia 12 5.00 Very Low Very Low Very Low
long-term potentiation 11 5.00 Very Low Very Low Very Low
Hippocampus 8 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
Dopamine 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 143 99.96 Very High Very High Very High
Aging 16 97.64 Very High Very High Very High
Apoptosis 6 73.12 Quite High
Disease 34 48.64 Quite Low
Death 13 43.80 Quite Low
Attention Deficit Hyperactivity Disorder 2 23.72 Low Low
Motor Neuron Diseases 6 17.16 Low Low
Neurodegenerative Disease 13 5.00 Very Low Very Low Very Low
Cv Unclassified Under Development 11 5.00 Very Low Very Low Very Low
Parkinson's Disease 10 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
During OS, ROS/RNS usually activate Ca2+ channels and repress Ca2+ pumps (Ermak and Davies, 2002), resulting in elevation of [Ca2+]i and initiation of downstream events mentioned above.
ROS Negative_regulation (repress) of Ca2 associated with stress
1) Confidence 0.19 Published 2010 Journal Frontiers in Aging Neuroscience Section Body Doc Link PMC2874397 Disease Relevance 0.61 Pain Relevance 0.04

General Comments

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