INT311659

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Context Info
Confidence 0.08
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.18
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Egfr, Mapk1) cytoplasm (Egfr, Mapk1) signal transduction (Egfr, Mapk1)
kinase activity (Egfr, Mapk1) nucleoplasm (Mapk1) mitochondrion (Mapk1)
Egfr (Mus musculus)
Mapk1 (Mus musculus)
Pain Link Frequency Relevance Heat
midbrain 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
vagus nerve 1 5.00 Very Low Very Low Very Low
isoflurane 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Congenital Anomalies 7 73.72 Quite High
Syndrome 1 52.00 Quite High
Craniosynostosis 1 51.60 Quite High
Polydactyly 1 41.04 Quite Low
Dwarfism 1 30.56 Quite Low
Cleft Palate 8 19.84 Low Low
Targeted Disruption 27 6.56 Low Low
Apoptosis 19 5.00 Very Low Very Low Very Low
Persistent Truncus Arteriosus 4 5.00 Very Low Very Low Very Low
Ventricular Heart Septal Defects 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
For example, Spry2 can potentiate EGFR signaling by binding to c-Cbl and sequestering it away from the EGFR, thus preventing EGFR down regulation and degradation, consequently leading to sustained EGFR activation, and enhanced ERK signaling.
EGFR Positive_regulation (enhanced) of ERK
1) Confidence 0.08 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2874773 Disease Relevance 0.18 Pain Relevance 0

General Comments

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