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Context Info
Confidence 0.01
First Reported 1987
Last Reported 1987
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.35
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Adra2a, Gh) plasma membrane (Adra2a, Gh) mitochondrion (Gh)
cytoplasm (Adra2a) signal transducer activity (Adra2a) cytosol (Gh)
Adra2a (Mus musculus)
Gh (Mus musculus)
Pain Link Frequency Relevance Heat
Clonidine 3 97.84 Very High Very High Very High
agonist 2 75.00 Quite High
Disease Link Frequency Relevance Heat
Endometriosis (extended) 2 99.00 Very High Very High Very High
Increased Venous Pressure Under Development 2 98.56 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results suggest that estrogens do not enhance alpha 2-adrenergic mechanisms that regulate GH secretion and that improvement in menopausal flushing after estrogen therapy is not mediated by an effect on central alpha 2-adrenergic function.
alpha 2-adrenergic Regulation (regulate) of Localization (secretion) of GH associated with endometriosis (extended) and increased venous pressure under development
1) Confidence 0.01 Published 1987 Journal J. Clin. Endocrinol. Metab. Section Abstract Doc Link 2884230 Disease Relevance 0.35 Pain Relevance 0.32

General Comments

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