INT312042

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.60
Pain Relevance 0.26

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (JUN) nucleoplasm (JUN) nuclear chromosome (JUN)
aging (JUN) nucleus (JUN) DNA binding (JUN)
Anatomy Link Frequency
joints 2
GLC1N (Homo sapiens)
JUN (Homo sapiens)
Pain Link Frequency Relevance Heat
Osteoarthritis 17 93.44 High High
Inflammation 5 28.92 Quite Low
cINOD 4 5.00 Very Low Very Low Very Low
cytokine 3 5.00 Very Low Very Low Very Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
Analgesic 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Osteoarthritis 17 93.44 High High
Death 1 78.72 Quite High
Cancer 4 31.64 Quite Low
INFLAMMATION 6 28.92 Quite Low
Necrosis 3 5.00 Very Low Very Low Very Low
Chondrosarcoma 3 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Aging 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low
Rheumatic Diseases 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We have previously shown that GlcN and NAPA were both effective in restoring normal cartilage morphology in injured rabbit joints and that GlcN can inhibit AP-1 activation by inhibiting MAP kinase phosphorylation.
GlcN Negative_regulation (inhibit) of Positive_regulation (activation) of AP-1 in joints
1) Confidence 0.01 Published 2010 Journal Arthritis Res Ther Section Body Doc Link PMC2875647 Disease Relevance 0.60 Pain Relevance 0.26

General Comments

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