INT313993

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.61
Pain Relevance 0.68

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Ros1) mitochondrion (Nfkb1) DNA binding (Nfkb1)
protein complex (Nfkb1) cytoplasm (Nfkb1) cell proliferation (Ros1)
Ros1 (Rattus norvegicus)
Nfkb1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 4 97.64 Very High Very High Very High
Glutamate 44 90.80 High High
Central nervous system 12 77.16 Quite High
Glutamate receptor 6 71.40 Quite High
Neurotransmitter 2 45.76 Quite Low
cerebral cortex 12 5.00 Very Low Very Low Very Low
Inflammation 6 5.00 Very Low Very Low Very Low
nMDA receptor 6 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 8 100.00 Very High Very High Very High
Cv Unclassified Under Development 4 97.64 Very High Very High Very High
Neurodegenerative Disease 4 94.00 High High
Stress 18 83.04 Quite High
Injury 6 74.92 Quite High
Hypoxia 126 74.64 Quite High
Disease 4 50.80 Quite High
Neurological Disease 6 5.00 Very Low Very Low Very Low
INFLAMMATION 6 5.00 Very Low Very Low Very Low
Reperfusion Injury 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Intracellular ROS are capable of inducing damage and, in severe cases, cell death through mitochondrial alterations leading to the release of cytochrome c 11-12, through activation of the JNK pathway 13 or by activation of nuclear factor-KB (NF-KB) transcription factors 14.
ROS Positive_regulation (activation) of NF-KB associated with death
1) Confidence 0.05 Published 2010 Journal International Journal of Medical Sciences Section Body Doc Link PMC2880843 Disease Relevance 0.81 Pain Relevance 0.34
Intracellular ROS are capable of inducing damage and, in severe cases, cell death through mitochondrial alterations leading to the release of cytochrome c 11-12, through activation of the JNK pathway 13 or by activation of nuclear factor-KB (NF-KB) transcription factors 14.
ROS Positive_regulation (activation) of factor-KB associated with death
2) Confidence 0.04 Published 2010 Journal International Journal of Medical Sciences Section Body Doc Link PMC2880843 Disease Relevance 0.81 Pain Relevance 0.34

General Comments

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