INT31450
From wiki-pain
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Sentences Mentioned In
Key: | Protein | Mutation | Event | Anatomy | Negation | Speculation | Pain term | Disease term |
Antihistamines act by competing with histamine for H1 or H2 histamine receptors on cell membranes. | |||||||||||||||
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The relative noradrenaline/serotonin reuptake inhibition, lipid solubility and their potency at histamine H1, muscarinic and alpha 1-adrenergic receptors had no substantial association with the FTI. | |||||||||||||||
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In vitro antagonism of benzodiazepine binding to cerebral receptors by H1 and H2 antihistamines. | |||||||||||||||
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It also has additional antagonistic affinity for the histaminic H1 and adrenergic ? | |||||||||||||||
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In fact, for some agents (eg, quetiapine, clozapine) the affinity for the H1 and ? | |||||||||||||||
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Even though we only had 50% power, we detected an association of H1 at a significance level of 0.001, and this association would survive a Bonferroni correction for the seventeen crude or adjusted association tests performed for haplogroups, suggesting that it is an important association. | |||||||||||||||
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Stratification by sex was performed as there are clear differences between male and female ischemic patients [33], and some of the associations (e.g. adjusted association of H1 in females) most likely did not reach statistical significance due to the relatively small sample sizes. | |||||||||||||||
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Reboxetine is a potent, selective, and specific noradrenaline reuptake inhibitor, with negligible affinity for muscarinic, H1, ? | |||||||||||||||
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SNPs m.3010G>A, m.7028C>T and m.11719G>A, which together define H1, were found to consistently influence the risk for ischemic stroke in both the uncorrected ? | |||||||||||||||
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We found suggestive evidence for association of the mitochondrial haplogroup H1 with ischemic stroke. | |||||||||||||||
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Stratification by sex revealed that the crude association of haplogroup H1 is quite consistent among males (OR = 0.65, 95%CI = 0.430.98, p = 0.038) and females (OR = 0.58, 95%CI = 0.370.93, p = 0.021), even though it did not reach significance in females when adjusted for co-variates (Figure 2), most likely due to the small sample size. | |||||||||||||||
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Several reports on the treatment of anaphylaxis have demonstrated that a combination of H1 and H2 antagonists is more effective than treatment with an H1 antagonist alone [24]. | |||||||||||||||
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0.02, H1 p? | |||||||||||||||
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Predominantly human subtypes (H1, H2, and H3) bind to alpha-2,6-galactose sialic acid found in ciliated human respiratory tract epithelium [18]. | |||||||||||||||
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The tagging SNPs for H1 were individually associated with higher CRP levels. | |||||||||||||||
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For comparison, H1 and H5, which included the triallelic proxy, were both associated with higher CRP levels. | |||||||||||||||
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Ki (affinity) values of the olopatadine hydrochloride binding to H1, H2, and H3 receptors were 4.11 × 10? | |||||||||||||||
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CR6261 has been shown to recognize H1 viruses that have emerged over a time span of 90 years from the H1N1 virus which caused the 1918 Spanish flu pandemic to the latest Brisbane viruses. | |||||||||||||||
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Affinity for H1, H2, and H3 receptors was examined using pyrilamine, tiotidine, and methylhistamine as the ligand, respectively. | |||||||||||||||
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It also binds with high affinity to H1 and M1 receptors. | |||||||||||||||
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General Comments
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