INT314729

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Context Info
Confidence 0.30
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 2.50
Pain Relevance 0.25

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (TP53) nucleoplasm (TP53) mitochondrion (TP53)
endoplasmic reticulum (TP53) enzyme binding (TP53) protein complex assembly (TP53)
TP53 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 10 74.88 Quite High
palliative 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 182 100.00 Very High Very High Very High
Breast Cancer 246 96.28 Very High Very High Very High
Targeted Disruption 12 89.68 High High
Inflammatory Breast Neoplasms 6 75.44 Quite High
Disease 32 64.52 Quite High
INFLAMMATION 4 61.00 Quite High
Metastasis 20 22.48 Low Low
Recurrence 18 5.00 Very Low Very Low Very Low
Carcinoma 14 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Among molecules regulating the p53 and RB tumor suppressor pathways, mutation of p53, overexpression of cyclin D1b, LMW cyclin E are apparently associated with poor clinical outcome of patients.
Regulation (regulating) of Localization (mutation) of p53 associated with cancer
1) Confidence 0.30 Published 2010 Journal Clinical Medicine Insights. Oncology Section Body Doc Link PMC2883240 Disease Relevance 1.25 Pain Relevance 0.13
Among molecules regulating the p53 and RB tumor suppressor pathways, mutation of p53, overexpression of cyclin D1b, LMW cyclin E are apparently associated with poor clinical outcome of patients.
Regulation (regulating) of Localization (mutation) of p53 associated with cancer
2) Confidence 0.30 Published 2010 Journal Clinical Medicine Insights. Oncology Section Body Doc Link PMC2883240 Disease Relevance 1.25 Pain Relevance 0.13

General Comments

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