INT314741

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Context Info
Confidence 0.36
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.66
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (VEGFA, Akt1) transport (Akt1) aging (Akt1)
enzyme binding (Akt1) carbohydrate metabolic process (Akt1) cytosol (Akt1)
VEGFA (Homo sapiens)
Akt1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 5 5.00 Very Low Very Low Very Low
palliative 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Breast Cancer 123 82.44 Quite High
Cancer 91 70.88 Quite High
Metastasis 10 69.52 Quite High
Recurrence 9 68.84 Quite High
Solid Tumor 1 67.20 Quite High
Disease 16 59.84 Quite High
Ductal Carcinoma 2 33.76 Quite Low
Lobular Carcinoma 1 27.20 Quite Low
Carcinoma 7 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Vascular endothelial growth factor (VEGF) is composed of a family of five isoforms (VEGFA, VEGFB, VEGFC, VEGFD, and PLGF) which act as ligands for tyrosine kinase receptors (VEGF-Rs).203 Upon binding of VEGF to its receptors (primarily VEGFR2), intracellular signaling pathways, including MEK-ERK and PI3K-Akt, are activated that mediate angiogenic switches (Fig. 1).
VEGF Binding (binding) of Akt
1) Confidence 0.36 Published 2010 Journal Clinical Medicine Insights. Oncology Section Body Doc Link PMC2883240 Disease Relevance 0.66 Pain Relevance 0

General Comments

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