INT315008

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Context Info
Confidence 0.31
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.73
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Cacna1d) plasma membrane (Cacna1d) transmembrane transport (Cacna1d)
Anatomy Link Frequency
neurons 2
Cacna1d (Mus musculus)
Pain Link Frequency Relevance Heat
Intracerebroventricular 1 78.76 Quite High
Neurotransmitter 7 23.44 Low Low
Glutamate 4 5.00 Very Low Very Low Very Low
addiction 3 5.00 Very Low Very Low Very Low
Action potential 3 5.00 Very Low Very Low Very Low
long-term potentiation 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
amygdala 1 5.00 Very Low Very Low Very Low
halothane 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Dystonia 10 98.52 Very High Very High Very High
Ataxia 2 81.68 Quite High
Stress 2 76.44 Quite High
Absence Epilepsy 1 63.00 Quite High
Disease 14 56.32 Quite High
Channelopathies 17 50.00 Quite Low
Anxiety Disorder 2 39.20 Quite Low
Hypokalemic Periodic Paralysis 29 5.00 Very Low Very Low Very Low
Night Blindness 9 5.00 Very Low Very Low Very Low
Malignant Hyperthermia 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This finding is surprising because LTCC expression in these neurons is very low, thus mediating only about 7% of the total Ca2+ channel current [21, 38].
Negative_regulation (low) of Gene_expression (expression) of LTCC in neurons
1) Confidence 0.31 Published 2010 Journal Pflugers Arch Section Body Doc Link PMC2883925 Disease Relevance 0.73 Pain Relevance 0.04

General Comments

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