INT315029

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Context Info
Confidence 0.04
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Cacna1f) cell cycle (Calm3)
Calm3 (Mus musculus)
Cacna1f (Mus musculus)
Pain Link Frequency Relevance Heat
Neurotransmitter 7 5.00 Very Low Very Low Very Low
Glutamate 4 5.00 Very Low Very Low Very Low
addiction 3 5.00 Very Low Very Low Very Low
Action potential 3 5.00 Very Low Very Low Very Low
long-term potentiation 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
amygdala 1 5.00 Very Low Very Low Very Low
Intracerebroventricular 1 5.00 Very Low Very Low Very Low
halothane 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Channelopathies 17 50.00 Quite Low
Calcification 1 12.28 Low Low
Hypokalemic Periodic Paralysis 29 5.00 Very Low Very Low Very Low
Disease 14 5.00 Very Low Very Low Very Low
Dystonia 10 5.00 Very Low Very Low Very Low
Night Blindness 9 5.00 Very Low Very Low Very Low
Malignant Hyperthermia 8 5.00 Very Low Very Low Very Low
Syndrome 6 5.00 Very Low Very Low Very Low
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Anxiety Disorder 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition, Singh et al. imply a role of the post-IQ motif for voltage-dependent inactivation [67]. b As shown in FRET experiments [70], the Cav1.4 CTM interferes with CaM binding to one or more sites responsible for CaM pre-association (apo-CaM) in intact cells.
CaM Binding (binding) of Cav1.4
1) Confidence 0.04 Published 2010 Journal Pflugers Arch Section Body Doc Link PMC2883925 Disease Relevance 0 Pain Relevance 0

General Comments

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