INT315418

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.63
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Slc26a5) plasma membrane (Slc26a5) transmembrane transport (Slc26a5)
Anatomy Link Frequency
neuronal 1
Slc26a5 (Mus musculus)
Pain Link Frequency Relevance Heat
Lasting pain 2 25.48 Quite Low
Central nervous system 2 5.00 Very Low Very Low Very Low
midbrain 2 5.00 Very Low Very Low Very Low
Inflammation 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
Bioavailability 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Infection 20 92.96 High High
Sprains And Strains 8 82.96 Quite High
Scrapie 10 81.04 Quite High
Chronic Wasting Disease 12 78.80 Quite High
Encephalitis 2 75.84 Quite High
Neurodegenerative Disease 4 67.60 Quite High
Rabies Virus Infection 6 61.16 Quite High
Prion Diseases 32 37.60 Quite Low
Targeted Disruption 8 27.40 Quite Low
Pain 2 25.48 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the present study, we demonstrate the ability of cationic liposomes to protect siRNA from serum degradation and of the RVG-9r peptide to specifically target these liposome-siRNA-peptide complexes (LSPCs) to neuronal cells, knockdown PrPC expression and greatly decrease PrPRES in chronically prion-infected neuronal cells in vitro.
Negative_regulation (decrease) of PrPRES in neuronal
1) Confidence 0.01 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2885418 Disease Relevance 0.20 Pain Relevance 0
PrP LSPCs eliminate PrPRES in vitro
Negative_regulation (eliminate) of PrPRES
2) Confidence 0.01 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2885418 Disease Relevance 0.42 Pain Relevance 0

General Comments

This test has worked.

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