INT316200

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.43
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.05
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Mc4r) plasma membrane (Mc4r) signal transducer activity (Mc4r)
Mc4r (Mus musculus)
Pain Link Frequency Relevance Heat
Neuropeptide 2 64.16 Quite High
cytokine 13 5.00 Very Low Very Low Very Low
Inflammation 5 5.00 Very Low Very Low Very Low
antagonist 3 5.00 Very Low Very Low Very Low
Buprenorphine 2 5.00 Very Low Very Low Very Low
analgesia 2 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Appetite Loss 45 99.54 Very High Very High Very High
Chronic Disease 4 96.36 Very High Very High Very High
Myocardial Infarction 104 90.00 High High
Frailty 1 89.16 High High
Weight Loss 3 86.64 High High
Congenital Anomalies 1 84.40 Quite High
Body Weight 11 41.28 Quite Low
Pulmonary Edema 6 37.64 Quite Low
Cancer 5 5.00 Very Low Very Low Very Low
Weight Gain 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results argue that in addition to attenuating cachexia in the acute illness, loss of MC4R signaling is also effective in providing prolonged protection against the development of cachexia in the setting of a chronic illness.
Regulation (effective) of Negative_regulation (loss) of MC4R associated with appetite loss and chronic disease
1) Confidence 0.43 Published 2010 Journal The Journal of Endocrinology Section Body Doc Link PMC2887273 Disease Relevance 1.05 Pain Relevance 0.03

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox