INT316230

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Context Info
Confidence 0.21
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.92
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endosome (BDKRB2) plasma membrane (BDKRB2) signal transducer activity (BDKRB2)
BDKRB2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Angina 2 99.84 Very High Very High Very High
bradykinin 41 85.12 High High
Inflammation 8 5.00 Very Low Very Low Very Low
b2 receptor 4 5.00 Very Low Very Low Very Low
antagonist 4 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cv General 3 Under Development 2 99.84 Very High Very High Very High
Myocardial Infarction 2 99.50 Very High Very High Very High
Diabetes Mellitus 69 95.80 Very High Very High Very High
Disease 3 78.40 Quite High
Increased Venous Pressure Under Development 3 75.24 Quite High
Atherosclerosis 4 6.72 Low Low
INFLAMMATION 9 5.00 Very Low Very Low Very Low
Stress 5 5.00 Very Low Very Low Very Low
Ocular Toxicity (including Many Sub-types) 3 5.00 Very Low Very Low Very Low
Diabetic Retinopathy 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In contrast to our earlier study, where patients with acute myocardial infarction or stable angina showed diminished abundance of B2R on CPCs, we could not detect any significant difference in kinin-receptor expression between cells from T1D and H donors [11].
Negative_regulation (diminished) of Localization (abundance) of B2R associated with angina, diabetes mellitus and myocardial infarction
1) Confidence 0.21 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2887352 Disease Relevance 0.92 Pain Relevance 0.21

General Comments

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