INT317318

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.83
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Gfm1, Trap1) unfolded protein binding (Trap1) protein folding (Trap1)
intracellular (Gfm1) translation (Gfm1) GTPase activity (Gfm1)
Gfm1 (Mus musculus)
Trap1 (Mus musculus)
Pain Link Frequency Relevance Heat
Dopamine 11 5.00 Very Low Very Low Very Low
Central nervous system 3 5.00 Very Low Very Low Very Low
midbrain 2 5.00 Very Low Very Low Very Low
Catecholamine 2 5.00 Very Low Very Low Very Low
Glutamate 2 5.00 Very Low Very Low Very Low
Locus ceruleus 1 5.00 Very Low Very Low Very Low
gABA 1 5.00 Very Low Very Low Very Low
monoamine 1 5.00 Very Low Very Low Very Low
Substantia nigra 1 5.00 Very Low Very Low Very Low
Eae 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Necrosis 1 100.00 Very High Very High Very High
Shock 1 100.00 Very High Very High Very High
Cancer 1 100.00 Very High Very High Very High
Apoptosis 7 99.92 Very High Very High Very High
Stress 17 99.56 Very High Very High Very High
Targeted Disruption 6 86.44 High High
Disease 88 77.08 Quite High
Parkinson's Disease 49 61.04 Quite High
Death 14 40.60 Quite Low
Drug Induced Neurotoxicity 2 33.92 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Moreover, Deas et al. review that PINK1 protects cells against oxidative stress induced apoptosis by interacting with the mitochondrial chaperone tumor necrosis factor receptor-associated protein 1 (TRAP1)/heat shock protein 75 (Hsp75) (Pridgeon et al., 2007).
mitochondrial Binding (interacting) of Hsp75 associated with stress, necrosis, cancer, shock and apoptosis
1) Confidence 0.01 Published 2010 Journal Frontiers in Aging Neuroscience Section Body Doc Link PMC2890153 Disease Relevance 0.83 Pain Relevance 0

General Comments

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