INT317388

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Context Info
Confidence 0.23
First Reported 2010
Last Reported 2010
Negated 0
Speculated 2
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.11
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

protein complex (Stk11, Prkag1) nucleus (Stk11, Prkag1) mitochondrion (Stk11)
response to stress (Prkag1) cytoplasm (Stk11) cytosol (Stk11)
Stk11 (Mus musculus)
Prkag1 (Mus musculus)
Pain Link Frequency Relevance Heat
ischemia 6 59.28 Quite High
alcohol 24 50.00 Quite Low
tolerance 12 45.60 Quite Low
ketamine 6 5.00 Very Low Very Low Very Low
anesthesia 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 6 59.28 Quite High
Obesity 4 55.20 Quite High
Coronary Heart Disease 8 50.00 Quite Low
Impaired Glucose Tolerance 20 45.60 Quite Low
Stress 8 40.96 Quite Low
Apoptosis 4 40.08 Quite Low
Targeted Disruption 10 34.08 Quite Low
Toxicity 14 5.00 Very Low Very Low Very Low
Alcoholic Cardiomyopathy 8 5.00 Very Low Very Low Very Low
Diabetes Mellitus 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Result from our current study revealed that acute ethanol treatment turned on LKB1 phosphorylation with a further increase in ADH mice, supporting a likely role of LKB1 in the ADH and ethanol-induced AMPK activation (shown by phosphorylation of AMPK and ACC).
LKB1 Spec (likely) Regulation (role) of Positive_regulation (activation) of AMPK
1) Confidence 0.23 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2890411 Disease Relevance 0.11 Pain Relevance 0.03
We went on to examine the involvement of LKB1 in ADH and ethanol-induced response in AMPK/ACC activation.
LKB1 Spec (examine) Regulation (involvement) of Positive_regulation (activation) of AMPK
2) Confidence 0.14 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2890411 Disease Relevance 0 Pain Relevance 0

General Comments

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