INT317553

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Context Info
Confidence 0.07
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 1.14

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Car2, Grin1) cytosol (Car2) transport (Grin1)
extracellular space (Car2) lyase activity (Car2) plasma membrane (Grin1)
Car2 (Mus musculus)
Grin1 (Mus musculus)
Pain Link Frequency Relevance Heat
opioid receptor 84 99.84 Very High Very High Very High
Kinase C 39 92.88 High High
Morphine 77 90.08 High High
tolerance 28 89.36 High High
addiction 1 60.56 Quite High
Calcium channel 1 51.84 Quite High
Opioid 14 50.00 Quite Low
nMDA receptor 1 49.80 Quite Low
Glutamate 10 44.56 Quite Low
Analgesic 19 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Urological Neuroanatomy 18 5.00 Very Low Very Low Very Low
Nociception 7 5.00 Very Low Very Low Very Low
Sprains And Strains 3 5.00 Very Low Very Low Very Low
Pain 3 5.00 Very Low Very Low Very Low
Intractable Pain 2 5.00 Very Low Very Low Very Low
Apoptosis 1 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low
Herpes Simplex Virus Infection 1 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low
Decapitation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
CaMKII, a Ca2+-CaM-activated protein kinase that is highly abundant in the CNS, has emerged as a critical factor in the NMDAR to MOR section of the regulatory loop.
Ca2 Binding (emerged) of NMDAR associated with opioid receptor
1) Confidence 0.07 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2890584 Disease Relevance 0 Pain Relevance 1.14

General Comments

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