INT320539

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Context Info
Confidence 0.26
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.46
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (JAK2) cytosol (JAK2) signal transduction (JAK2)
histone binding (JAK2) cytoskeleton (JAK2) nucleus (JAK2)
JAK2 (Homo sapiens)
Pain Link Frequency Relevance Heat
aspirin 3 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Angina 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Coronary Artery Disease 66 97.94 Very High Very High Very High
Apoptosis 5 71.92 Quite High
Stress 1 48.96 Quite Low
Rheumatoid Arthritis 19 15.76 Low Low
Atherosclerosis 13 5.00 Very Low Very Low Very Low
Adhesions 4 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 4 5.00 Very Low Very Low Very Low
Disease 4 5.00 Very Low Very Low Very Low
INFLAMMATION 3 5.00 Very Low Very Low Very Low
Myocardial Infarction 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
EPC from CAD patients were shown to have a reduced revascularization capacity, with diminished CXCR4 signaling, as Jak2 phosphorylation by CXCL12 is impaired, compared to EPC from healthy individuals, while the expression levels of CXCR4 are normal [24].
Negative_regulation (impaired) of Phosphorylation (phosphorylation) of Jak2 associated with coronary artery disease
1) Confidence 0.26 Published 2010 Journal BMC Genomics Section Body Doc Link PMC2901320 Disease Relevance 0.46 Pain Relevance 0

General Comments

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