INT321412

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.18
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (ABCC8, SELE) extracellular space (SELE) small molecule metabolic process (ABCC8)
cell adhesion (SELE) ATPase activity (ABCC8) carbohydrate metabolic process (ABCC8)
ABCC8 (Homo sapiens)
SELE (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 29 58.96 Quite High
cytokine 13 33.12 Quite Low
Kinase C 10 5.00 Very Low Very Low Very Low
bradykinin 3 5.00 Very Low Very Low Very Low
spinal inflammation 2 5.00 Very Low Very Low Very Low
COX2 1 5.00 Very Low Very Low Very Low
COX-2 inhibitor 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Bioavailability 1 5.00 Very Low Very Low Very Low
Etanercept 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperinsulinism 8 100.00 Very High Very High Very High
Obesity 8 81.44 Quite High
Insulin Resistance 27 80.44 Quite High
Hypertension 10 79.20 Quite High
Adhesions 16 77.64 Quite High
Diabetes Mellitus 83 65.68 Quite High
INFLAMMATION 29 58.96 Quite High
Stress 31 58.28 Quite High
Apoptosis 89 50.00 Quite Low
Vasculitis 4 49.48 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Through activation of the MAP-kinase signaling pathways, hyperinsulinemia promotes secretion of ET-1, activates cation pumps, and increases expression of VCAM-1 and E-selectin [46].
hyperinsulinemia Positive_regulation (increases) of Gene_expression (expression) of E-selectin associated with hyperinsulinism
1) Confidence 0.00 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2903979 Disease Relevance 1.18 Pain Relevance 0.03

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox