INT321430

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Context Info
Confidence 0.20
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.00
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (FLT1) endosome (FLT1) Golgi apparatus (FLT1)
cytoplasm (FLT1) extracellular space (FLT1) extracellular region (FLT1)
FLT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cva 1 60.08 Quite High
Inflammation 29 50.00 Quite Low
cytokine 13 27.40 Quite Low
Kinase C 10 5.00 Very Low Very Low Very Low
bradykinin 3 5.00 Very Low Very Low Very Low
spinal inflammation 2 5.00 Very Low Very Low Very Low
COX2 1 5.00 Very Low Very Low Very Low
COX-2 inhibitor 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Bioavailability 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperglycemia 38 96.92 Very High Very High Very High
Increased Venous Pressure Under Development 19 92.08 High High
Diabetes Mellitus 83 87.60 High High
Atherosclerosis 2 76.00 Quite High
Cardiovascular Disease 8 64.16 Quite High
Cv General 3 Under Development 1 60.08 Quite High
Adhesions 16 50.16 Quite High
Apoptosis 89 50.00 Quite Low
INFLAMMATION 29 50.00 Quite Low
Toxicity 1 46.72 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A supportive finding for this hypothesis is the fact that VEGF receptor 1 (FLT1) and PAI-1, both known Egr-1 responsive genes, are also increased in the presence of glucose and insulin.
Positive_regulation (increased) of FLT1 Binding (receptor) of
1) Confidence 0.20 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2903979 Disease Relevance 1.00 Pain Relevance 0.03

General Comments

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