INT321431

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Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.77
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (DECR1) small molecule metabolic process (DECR1) nucleolus (DECR1)
nucleus (DECR1) cytoplasm (DECR1)
DECR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Kinase C 10 75.20 Quite High
Inflammation 29 50.00 Quite Low
cytokine 13 5.00 Very Low Very Low Very Low
bradykinin 3 5.00 Very Low Very Low Very Low
spinal inflammation 2 5.00 Very Low Very Low Very Low
COX2 1 5.00 Very Low Very Low Very Low
COX-2 inhibitor 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Bioavailability 1 5.00 Very Low Very Low Very Low
Etanercept 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 83 98.56 Very High Very High Very High
Stress 31 91.28 High High
Hyperglycemia 38 88.04 High High
Apoptosis 89 50.00 Quite Low
INFLAMMATION 29 50.00 Quite Low
Injury 5 49.84 Quite Low
Coronary Artery Disease 1 38.16 Quite Low
Increased Venous Pressure Under Development 19 37.08 Quite Low
Insulin Resistance 27 5.00 Very Low Very Low Very Low
Adhesions 16 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition to mitochondrial uncoupling there are other mechanisms that can contribute to superoxide production in diabetes, namely, uncoupling of eNOS, increased peroxidation and glycoxidation, activation of NADPH oxidases, decreased clearance of superoxide, and impaired antioxidant status [61].
Negative_regulation (decreased) of Positive_regulation (activation) of NADPH associated with diabetes mellitus
1) Confidence 0.03 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2903979 Disease Relevance 0.77 Pain Relevance 0.04

General Comments

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