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Context Info
Confidence 0.22
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.93
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Vdac1) mitochondrion (Vdac1) plasma membrane (Vdac1)
protein complex (Vdac1) transmembrane transport (Vdac1)
Anatomy Link Frequency
pore 2
Vdac1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory response 13 57.68 Quite High
Inflammation 33 25.48 Quite Low
anesthesia 1 5.00 Very Low Very Low Very Low
isoflurane 1 5.00 Very Low Very Low Very Low
cva 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 40 97.52 Very High Very High Very High
Apoptosis 18 94.60 High High
Targeted Disruption 2 89.76 High High
Stress 1 85.84 High High
Disease 8 74.68 Quite High
Infection 19 59.56 Quite High
INFLAMMATION 42 57.28 Quite High
Influenza Virus Infection 26 50.00 Quite Low
Sprains And Strains 74 44.08 Quite Low
Influenza In Birds 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
ANT3 and VDAC1 molecules are major components of the permeability transition pore of the mitochondrial membrane and enable cytochrome c release from the mitochondria, leading to the activation of the caspase pathway and subsequent cell death.
Positive_regulation (leading) of Localization (release) of VDAC1 in pore associated with death
1) Confidence 0.22 Published 2010 Journal PLoS Pathogens Section Body Doc Link PMC2908617 Disease Relevance 0.93 Pain Relevance 0.03

General Comments

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