INT322705

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Context Info
Confidence 0.08
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.77
Pain Relevance 0.65

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Cxcl2) extracellular region (Cxcl2)
Anatomy Link Frequency
Th1 cells 1
Cxcl2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
chemokine 56 100.00 Very High Very High Very High
antagonist 18 100.00 Very High Very High Very High
Inflammation 34 81.20 Quite High
Inflammatory response 2 69.84 Quite High
Intracerebroventricular 2 65.52 Quite High
ischemia 6 56.88 Quite High
anesthesia 2 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
medulla 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Graft Vs Host Disease 2 86.28 High High
Renal Disease 4 85.68 High High
INFLAMMATION 36 81.20 Quite High
Injury 18 79.64 Quite High
Cv General 4 Under Development 2 64.12 Quite High
Cv Unclassified Under Development 6 56.88 Quite High
Vascular System Injuries 12 5.00 Very Low Very Low Very Low
Necrosis 4 5.00 Very Low Very Low Very Low
Herpes Simplex Virus 4 5.00 Very Low Very Low Very Low
Thrombosis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This shows that vMIP-II, as a peptide chemokine receptor antagonist, without a degradation inhibiting protection, is not potent enough to fully prevent acute rejection-induced damage and perfusion disturbance.
Protein_catabolism (degradation) of vMIP-II associated with chemokine and antagonist
1) Confidence 0.08 Published 2010 Journal World J Urol Section Body Doc Link PMC2908759 Disease Relevance 0.24 Pain Relevance 0.40
and lymphotactin, as well as the chemokine receptors CCR2 and CCR5, in the absence of upregulation of CCR3 and CCR8, which are Th2 chemokine receptor phenotypes [19]. vMIP-II inhibited the firm arrest and transmigration of Th1 cells under in vitro flow conditions and exerted an agonistic action on CCR3 driving a Th2 immune response [16].
Protein_catabolism (phenotypes) of vMIP-II in Th1 cells associated with chemokine
2) Confidence 0.05 Published 2010 Journal World J Urol Section Body Doc Link PMC2908759 Disease Relevance 0.53 Pain Relevance 0.25

General Comments

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