INT322848

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Context Info
Confidence 0.09
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (PIK3CD) signal transduction (PIK3CD) extracellular space (VEGFA)
growth (VEGFA) extracellular region (VEGFA) proteinaceous extracellular matrix (VEGFA)
VEGFA (Homo sapiens)
PIK3CD (Homo sapiens)
Pain Link Frequency Relevance Heat
Central nervous system 4 5.00 Very Low Very Low Very Low
Glutamate 4 5.00 Very Low Very Low Very Low
imagery 4 5.00 Very Low Very Low Very Low
withdrawal 2 5.00 Very Low Very Low Very Low
headache 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 18 50.00 Quite Low
Stroke 4 5.00 Very Low Very Low Very Low
Metastasis 2 5.00 Very Low Very Low Very Low
Aids-related Complex 2 5.00 Very Low Very Low Very Low
Congenital Anomalies 2 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Headache 2 5.00 Very Low Very Low Very Low
Increased Venous Pressure Under Development 2 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Convulsion 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
PI3K can be activated by several growth factors, including VEGF.
VEGF Positive_regulation (activated) of PI3K
1) Confidence 0.09 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2909203 Disease Relevance 0 Pain Relevance 0
Characterizing the role of PDCD10 during PI3K activation by VEGF, as well as downstream effectors such as AKT1, will be important in understanding CCM development and how we may treat this condition by modulating the activity of these kinases.
VEGF Positive_regulation (activation) of PI3K
2) Confidence 0.09 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2909203 Disease Relevance 0 Pain Relevance 0

General Comments

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