INT323279

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.67
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Nos3, Arg2) Golgi apparatus (Nos3) cytoplasm (Nos3)
signal transduction (Nos3) oxidoreductase activity (Nos3) nucleolus (Nos3)
Nos3 (Mus musculus)
Arg2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
potassium channel 4 31.48 Quite Low
Calcium channel 2 20.40 Low Low
Bioavailability 4 19.52 Low Low
TRP channel 1 15.28 Low Low
Tetrahydrobiopterin 21 5.00 Very Low Very Low Very Low
adenocard 3 5.00 Very Low Very Low Very Low
antagonist 3 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
cOX1 2 5.00 Very Low Very Low Very Low
COX-2 inhibitor 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cardiovascular Disease 9 97.44 Very High Very High Very High
Stress 4 94.88 High High
Hypertension 45 75.92 Quite High
Atherosclerotic Plaque 1 67.28 Quite High
Hyperlipidemia 2 52.92 Quite High
Increased Venous Pressure Under Development 14 5.00 Very Low Very Low Very Low
Diabetes Mellitus 7 5.00 Very Low Very Low Very Low
Coronary Heart Disease 2 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low
Disease 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Arginase-2 competes with eNOS for substrate, and its expression and activity are enhanced in cardiovascular diseases, perhaps because of increased oxidative stress.
eNOS Binding (competes) of Arginase-2 associated with stress and cardiovascular disease
1) Confidence 0.01 Published 2010 Journal Curr Hypertens Rep Section Body Doc Link PMC2910890 Disease Relevance 0.67 Pain Relevance 0

General Comments

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