INT323465

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Context Info
Confidence 0.02
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.05
Pain Relevance 0.13

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Kcnj6) Golgi apparatus (Kcnj6) plasma membrane (Kcnj6)
cytoplasm (Kcnj6)
Kcnj6 (Mus musculus)
Pain Link Frequency Relevance Heat
Action potential 1 91.12 High High
Calcium channel 1 90.08 High High
antagonist 14 77.76 Quite High
Neurotransmitter 1 15.44 Low Low
tolerance 18 5.00 Very Low Very Low Very Low
Somatostatin 4 5.00 Very Low Very Low Very Low
Kinase C 3 5.00 Very Low Very Low Very Low
vagus nerve 2 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
Opioid 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 54 51.60 Quite High
Obesity 49 5.00 Very Low Very Low Very Low
Diabetes Mellitus 30 5.00 Very Low Very Low Very Low
Impaired Glucose Tolerance 20 5.00 Very Low Very Low Very Low
Insulin Resistance 18 5.00 Very Low Very Low Very Low
Hyperinsulinism 10 5.00 Very Low Very Low Very Low
Body Weight 9 5.00 Very Low Very Low Very Low
Disease 6 5.00 Very Low Very Low Very Low
Appetite Loss 5 5.00 Very Low Very Low Very Low
Hyperglycemia 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, an expanding bulk of data also makes it apparent that this KATP-channel dependent mechanism of glucose-stimulated insulin secretion does not fully describe the islet glucose response, and signals other than changes in ATP: ADP ratio have been increasingly implicated as important regulators of insulin secretion in recent years.
Regulation (regulators) of Localization (secretion) of KATP
1) Confidence 0.02 Published 2010 Journal International Journal of Peptides Section Body Doc Link PMC2911604 Disease Relevance 0.05 Pain Relevance 0.13

General Comments

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