INT324878

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.25
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Ncf1, CYBB) cytoplasm (Ncf1, CYBB) cell proliferation (Ncf1)
cytosol (Ncf1) mitochondrion (CYBB) oxidoreductase activity (CYBB)
Ncf1 (Mus musculus)
CYBB (Homo sapiens)
Pain Link Frequency Relevance Heat
cva 2 48.88 Quite Low
ischemia 4 16.04 Low Low
Inflammation 14 5.00 Very Low Very Low Very Low
Inflammatory response 7 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Inflammatory mediators 1 5.00 Very Low Very Low Very Low
Paracetamol 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hemorrhagic Shock 32 97.14 Very High Very High Very High
Hemorrhage 2 48.88 Quite Low
Injury 18 40.76 Quite Low
Shock 7 34.40 Quite Low
Lung Injury 1 20.44 Low Low
Cv Unclassified Under Development 4 16.04 Low Low
Adult Respiratory Distress Syndrome 27 5.00 Very Low Very Low Very Low
INFLAMMATION 15 5.00 Very Low Very Low Very Low
Systemic Inflammatory Response Syndrome 6 5.00 Very Low Very Low Very Low
Pneumonia 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It has been demonstrated that HS through HMGB1 activates the TLR4-MyD88-IRAK4 signaling pathway and further activates p38 MAPK and Akt pathways to initiate phosphorylation of p47phox and subsequent activation of NADPH oxidase (Figure 3) [2].
p47phox Positive_regulation (activation) of NADPH oxidase associated with hemorrhagic shock
1) Confidence 0.01 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2913855 Disease Relevance 0.25 Pain Relevance 0

General Comments

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