INT324886

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Context Info
Confidence 0.37
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.95
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endosome (Ece1) peptidase activity (Ece1) Golgi apparatus (Ece1)
plasma membrane (Ece1)
Anatomy Link Frequency
myocardium 1
Ece1 (Mus musculus)
Pain Link Frequency Relevance Heat
antagonist 11 99.66 Very High Very High Very High
cytokine 16 84.36 Quite High
Dismenorea 1 80.80 Quite High
Inflammatory response 5 71.04 Quite High
Inflammation 22 66.72 Quite High
fibrosis 20 52.24 Quite High
imagery 1 43.68 Quite Low
Central nervous system 1 7.48 Low Low
metalloproteinase 22 5.04 Very Low Very Low Very Low
ischemia 7 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Increased Venous Pressure Under Development 5 99.18 Very High Very High Very High
Sprains And Strains 11 98.60 Very High Very High Very High
Pre-term Labor 13 98.46 Very High Very High Very High
Infection 61 97.06 Very High Very High Very High
Heart Disease 12 92.64 High High
Adhesions 4 85.16 High High
Chagas Cardiomyopathy 34 83.28 Quite High
Dysmenorrhea 1 80.80 Quite High
Premature Birth 40 79.20 Quite High
Cardiomyopathy 25 75.12 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Subsequently, we have shown that ECE-1 is upregulated in our mouse model of PTL and that PTD is controlled with the ETA receptor antagonist BQ-123 and with silencing of ECE-1 mRNA [68] (Figure 2).
Transcription (silencing) of ECE-1 associated with antagonist and pre-term labor
1) Confidence 0.37 Published 2010 Journal Obstetrics and Gynecology International Section Body Doc Link PMC2913859 Disease Relevance 0.65 Pain Relevance 0.17
The infection of CD1 mice with the Brazil strain and C57BL/6 mice with the Tulahuen strain caused an intense vasculitis, high plasma ET-1 levels, and increased expression of mRNAs for the precursor molecule preproET-1, endothelin converting enzyme (ECE), and ET-1 in the myocardium [76].
Transcription (expression) of endothelin converting enzyme in myocardium associated with increased venous pressure under development, sprains and strains and infection
2) Confidence 0.09 Published 2010 Journal PLoS Neglected Tropical Diseases Section Body Doc Link PMC2930857 Disease Relevance 1.31 Pain Relevance 0.24

General Comments

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