INT325563

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Cacna1c) plasma membrane (Cacna1c) nucleus (Cacna1c)
transmembrane transport (Cacna1c) cytoplasm (Cacna1c)
Anatomy Link Frequency
myocytes 2
Cacna1c (Mus musculus)
Pain Link Frequency Relevance Heat
imagery 1 34.84 Quite Low
agonist 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Natriuresis 5 50.00 Quite Low
Coronary Heart Disease 13 5.00 Very Low Very Low Very Low
Body Weight 5 5.00 Very Low Very Low Very Low
Hypertrophy 4 5.00 Very Low Very Low Very Low
Hypertension 3 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 3 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Pressure Volume 2 Under Development 2 5.00 Very Low Very Low Very Low
Anaemia 1 5.00 Very Low Very Low Very Low
Fibrosis 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Pretreatment with ANP fully inhibited Ang II-stimulation of L-type Ca2+ channel activity (Fig. 2b) and had no effect on ISO-stimulation of L-type Ca2+ channel activity (Fig. 2d).Fig. 2L-type Ca2+ channel activity was analyzed in ventricular myocytes by whole-cell patch-clamp recordings.
Negative_regulation (inhibited) of Positive_regulation (stimulation) of L-type in myocytes
1) Confidence 0.05 Published 2010 Journal Basic Res Cardiol Section Body Doc Link PMC2916114 Disease Relevance 0 Pain Relevance 0

General Comments

This test has worked.

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