INT325568

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Context Info
Confidence 0.41
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.13
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Rgs2) plasma membrane (Rgs2) nucleus (Rgs2)
cell cycle (Rgs2) cytoplasm (Rgs2)
Anatomy Link Frequency
plasma 1
Rgs2 (Mus musculus)
Pain Link Frequency Relevance Heat
agonist 4 97.56 Very High Very High Very High
anesthesia 2 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Coronary Heart Disease 26 78.88 Quite High
Natriuresis 10 50.00 Quite Low
Body Weight 10 5.00 Very Low Very Low Very Low
Hypertrophy 8 5.00 Very Low Very Low Very Low
Hypertension 6 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 6 5.00 Very Low Very Low Very Low
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Pressure Volume 2 Under Development 4 5.00 Very Low Very Low Very Low
Anaemia 2 5.00 Very Low Very Low Very Low
Fibrosis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
As shown in Fig. 4, ANP (10 nM, incubation for 30, 60 and 120 min) caused a time-dependent increase in the total expression of RGS2 as well as in the amount of RGS2 associated with the plasma membrane.
Positive_regulation (increase) of RGS2 in plasma
1) Confidence 0.41 Published 2010 Journal Basic Res Cardiol Section Body Doc Link PMC2916114 Disease Relevance 0.05 Pain Relevance 0
We conclude that ANP, via GC-A/cGMP/PKG I signalling and activation of RGS2, specifically counter regulates the hypertrophic effects of agonists linked to G?
Positive_regulation (activation) of RGS2 associated with agonist
2) Confidence 0.38 Published 2010 Journal Basic Res Cardiol Section Body Doc Link PMC2916114 Disease Relevance 0.08 Pain Relevance 0.05

General Comments

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