INT325836

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.22
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

GTPase activity (Dnm2, Gnas) cytoplasm (Dnm2, Gnas) plasma membrane (Dnm2, Gnas)
endosome (Gnas) protein complex (Dnm2) signal transducer activity (Gnas)
Dnm2 (Mus musculus)
Gnas (Mus musculus)
Pain Link Frequency Relevance Heat
medulla 4 9.72 Low Low
GABAergic 14 5.00 Very Low Very Low Very Low
depression 8 5.00 Very Low Very Low Very Low
Pyramidal cell 8 5.00 Very Low Very Low Very Low
Neurotransmitter 4 5.00 Very Low Very Low Very Low
Somatosensory cortex 4 5.00 Very Low Very Low Very Low
tetrodotoxin 2 5.00 Very Low Very Low Very Low
cerebral cortex 2 5.00 Very Low Very Low Very Low
ketamine 2 5.00 Very Low Very Low Very Low
Neuronal excitability 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ataxia 14 55.40 Quite High
Convulsion 68 53.96 Quite High
Epilepsy 30 50.00 Quite Low
Hearing Impairment 8 42.60 Quite Low
Ganglion Cysts 2 33.20 Quite Low
Sprains And Strains 12 15.28 Low Low
Depression 8 5.00 Very Low Very Low Very Low
Neurological Disease 8 5.00 Very Low Very Low Very Low
Congenital Anomalies 8 5.00 Very Low Very Low Very Low
Disease 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This particular alternative exon pair of Dnm1 and Dnm2 is conserved in all vertebrates including mice (e.g.
Dnm2 Binding (pair) of exon
1) Confidence 0.00 Published 2010 Journal PLoS Genetics Section Body Doc Link PMC2916854 Disease Relevance 0.11 Pain Relevance 0
This particular alternative exon pair of Dnm1 and Dnm2 is conserved in all vertebrates including mice (e.g.
Dnm2 Binding (pair) of exon
2) Confidence 0.00 Published 2010 Journal PLoS Genetics Section Body Doc Link PMC2916854 Disease Relevance 0.11 Pain Relevance 0

General Comments

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