INT32586

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Context Info
Confidence 0.03
First Reported 1988
Last Reported 1988
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.14
Pain Relevance 2.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (H2-Ab1) intracellular (H2-Ab1) cell-cell signaling (Pomc)
cytoplasm (Pomc) signal transduction (Pomc) extracellular space (Pomc)
H2-Ab1 (Mus musculus)
Pomc (Mus musculus)
Pain Link Frequency Relevance Heat
Morphine 12 100.00 Very High Very High Very High
analgesia 8 100.00 Very High Very High Very High
intrathecal 3 95.56 Very High Very High Very High
tail-flick 1 94.08 High High
Analgesic 1 85.12 High High
Intracerebroventricular 1 75.00 Quite High
opioid receptor 1 74.88 Quite High
Opioid 1 70.40 Quite High
Pain 2 69.24 Quite High
Disease Link Frequency Relevance Heat
Pain 2 69.24 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
On the other hand, i.t. beta-FNA blocked both i.t. beta-endorphin- and morphine-induced analgesia, but was more effective in blocking the effects of i.t. morphine than beta-endorphin.
beta-FNA Regulation (effective) of beta-endorphin associated with analgesia and morphine
1) Confidence 0.03 Published 1988 Journal J. Pharmacol. Exp. Ther. Section Abstract Doc Link 2966856 Disease Relevance 0.14 Pain Relevance 2.19

General Comments

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