INT325903

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Context Info
Confidence 0.02
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.26
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Gopc) peptidase activity (F2) extracellular space (F2)
extracellular region (F2) Golgi apparatus (Gopc) plasma membrane (Gopc)
F2 (Mus musculus)
Gopc (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 4 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Arthritis 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Coagulation Disorder 36 73.24 Quite High
Disease 26 50.00 Quite Low
Bacteremia 20 13.28 Low Low
Body Weight 6 8.72 Low Low
Abscess 126 5.00 Very Low Very Low Very Low
Sprains And Strains 50 5.00 Very Low Very Low Very Low
Staphylococcus Infection 30 5.00 Very Low Very Low Very Low
Infection 30 5.00 Very Low Very Low Very Low
Immunization 18 5.00 Very Low Very Low Very Low
Mrsa Infection 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Specific antibodies blocked the associations between vWbp and prothrombin or fibrinogen in a dose-dependent manner (Fig. 5C, D).
prothrombin Binding (associations) of Fig
1) Confidence 0.02 Published 2010 Journal PLoS Pathogens Section Body Doc Link PMC2916881 Disease Relevance 0.14 Pain Relevance 0
These findings corroborate results from the coagulation assays and demonstrate that specific polyclonal antibodies block the ability of Coa or vWbp to form active complexes with prothrombin that then act on fibrinogen (Fig. 3A).
prothrombin Binding (complexes) of Fig
2) Confidence 0.02 Published 2010 Journal PLoS Pathogens Section Body Doc Link PMC2916881 Disease Relevance 0.12 Pain Relevance 0

General Comments

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