INT326901

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Context Info
Confidence 0.28
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 0.46
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (PLD2, Pld1) endosome (Pld1) plasma membrane (PLD2)
nucleus (PLD2) cytoskeleton organization (PLD2)
PLD2 (Homo sapiens)
Pld1 (Mus musculus)
Pain Link Frequency Relevance Heat
agonist 3 22.88 Low Low
metalloproteinase 3 5.00 Very Low Very Low Very Low
cINOD 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Colon Cancer 99 83.12 Quite High
Familial Adenomatous Polyposis 9 69.52 Quite High
Cancer 72 67.88 Quite High
Breast Cancer 12 60.52 Quite High
Metastasis 6 5.00 Very Low Very Low Very Low
Sarcoma 3 5.00 Very Low Very Low Very Low
Pathologic Processes 3 5.00 Very Low Very Low Very Low
Glioma 3 5.00 Very Low Very Low Very Low
INFLAMMATION 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The polyclonal anti-PLD antibody that recognizes both PLD1 and PLD2 was generated as previously described [18].


PLD2 Binding (recognizes) of PLD1
1) Confidence 0.28 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2920823 Disease Relevance 0 Pain Relevance 0
LiCl was injected into mice, and expression of PLD was investigated in several tissues using anti-PLD antibody which recognizes both PLD1 and PLD2.
PLD2 Binding (recognizes) of PLD1
2) Confidence 0.28 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2920823 Disease Relevance 0.38 Pain Relevance 0
Anti-PLD antibody was generated against C-terminal peptide of PLD1 in which 7 amino acids among 12 amino acids were the same with that of PLD2, and recognizes both PLD1 and PLD2 [18].
PLD2 Binding (recognizes) of PLD1
3) Confidence 0.24 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2920823 Disease Relevance 0.08 Pain Relevance 0

General Comments

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