INT327365

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.21
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.68
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

unfolded protein binding (Hspa5) endoplasmic reticulum (Hspa5) nucleus (Hspa5)
response to stress (Hspa5)
Hspa5 (Mus musculus)
Pain Link Frequency Relevance Heat
metalloproteinase 5 42.80 Quite Low
Inflammation 9 37.28 Quite Low
Peripheral nervous system 2 5.00 Very Low Very Low Very Low
Spinal cord 1 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
Multiple sclerosis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 11 99.42 Very High Very High Very High
Familial Amyloidotic Polyneuropathy 21 89.64 High High
Apoptosis 4 69.52 Quite High
Death 4 52.32 Quite High
Alzheimer's Dementia 32 46.08 Quite Low
INFLAMMATION 9 37.28 Quite Low
Amyloidosis 1 32.36 Quite Low
Disease 7 23.04 Low Low
Asthma 2 22.00 Low Low
Amyloid Plaque 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The ability of TUDCA to abolish ER stress was previously observed and reduction of BiP induction and caspase 12 activation were reported [15]; TUDCA is also able to protect against oxidative stress in tissues damaged by TTR deposition and thus, TUDCA can act at different levels, affecting TTR extracellular deposition.
Negative_regulation (reduction) of Positive_regulation (induction) of BiP associated with stress
1) Confidence 0.21 Published 2010 Journal J Transl Med Section Body Doc Link PMC2922089 Disease Relevance 0.68 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox