INT327993

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.69
Pain Relevance 0.56

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endoplasmic reticulum (STT3A)
Anatomy Link Frequency
plasma 1
STT3A (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 6 88.32 High High
Analgesic 1 87.08 High High
c fibre 1 85.36 High High
qutenza 13 82.52 Quite High
agonist 8 82.08 Quite High
antagonist 11 78.36 Quite High
Inflammation 41 77.20 Quite High
Arthritis 10 58.88 Quite High
TRP channel 1 29.04 Quite Low
Inflammatory mediators 3 11.12 Low Low
Disease Link Frequency Relevance Heat
Stress 1 89.40 High High
Pain 7 88.32 High High
Injury 3 88.00 High High
Targeted Disruption 2 80.92 Quite High
INFLAMMATION 34 77.20 Quite High
Nociception 15 68.84 Quite High
Arthritis 22 58.88 Quite High
Hyperalgesia 6 5.00 Very Low Very Low Very Low
Neuropathic Pain 4 5.00 Very Low Very Low Very Low
Osteoarthritis 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Evidence supports the potential for ROS themselves to activate TRPV1 cation channels after conformational change of this integral membrane protein [12], directly increasing plasma membrane permeability to calcium ions [12,13].
Regulation (change) of integral membrane protein in plasma
1) Confidence 0.01 Published 2010 Journal Mol Pain Section Body Doc Link PMC2924298 Disease Relevance 0.69 Pain Relevance 0.56

General Comments

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