INT328606

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Context Info
Confidence 0.10
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.42

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Slc12a2) plasma membrane (Slc12a2) transmembrane transport (Slc12a2)
Slc12a2 (Mus musculus)
Pain Link Frequency Relevance Heat
gABA 16 98.92 Very High Very High Very High
GABAergic 13 90.32 High High
Pyramidal cell 41 80.24 Quite High
antagonist 1 72.44 Quite High
Action potential 2 51.04 Quite High
Hippocampus 23 12.72 Low Low
interneuron 5 5.00 Very Low Very Low Very Low
Cholecystokinin 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Epilepsy 3 5.00 Very Low Very Low Very Low
Convulsion 2 5.00 Very Low Very Low Very Low
Cognitive Disorder 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low
Frontal Lobe Epilepsy 1 5.00 Very Low Very Low Very Low
Brain Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The ontogenetic shift to hyperpolarizing GABA action is caused by a concomitant developmental down-regulation of NKCC1 and an up-regulation of a K+–Cl?
Regulation (regulation) of Regulation (regulation) of NKCC1 associated with gaba
1) Confidence 0.10 Published 2010 Journal Frontiers in Cellular Neuroscience Section Body Doc Link PMC2927192 Disease Relevance 0 Pain Relevance 0.42

General Comments

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